Theodore E Warkentin.
Theodore E Warkentin. Introduction: Devastating thromboses can complicate heparin-induced thrombocytopenia (HIT) and disseminated intravascular coagulation (DIC). A 64-year-old woman who is hospitalized with endocarditis and whose condition is clinically stable while she is receiving intravenous antibiotic agents has had a decrease in platelet count from 161,000 per cubic millimeter on day 7 of hospitalization to 60,000 per cubic millimeter on day 9. She has been receiving heparin at a dose of 40.
Heparin-induced thrombocytopenia (HIT) is an antibody-mediated adverse effect of heparin. This issue, pp 1486–91.
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ANDREAS GREINACHER is Director of the Department of Transfusion Medicine, Institute for Immunology and Transfusion Medicine,, Greifswald, Germany. Dr. Greinacher received the . degree from the Julius Maximilian University, Würzburg, Germany, and finished training in clinical immunology and transfusion medicine at the Justus-Liebig University, Giessen, Germany.
Heparin-induced thrombocytopenia (HIT) is the development of thrombocytopenia (a low platelet count), due to the administration of various forms of heparin, an anticoagulant. HIT predisposes to thrombosis (the abnormal formation of blood clots inside a blood vessel) because platelets release microparticles that activate thrombin, thereby leading to thrombosis. When thrombosis is identified the condition is called heparin-induced thrombocytopenia and thrombosis (HITT)
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Heparin-Induced Thrombocytopenia book.
Warkentin Heparin-Induced Thrombocytopenia in Children Anne F. Klenner, Andreas Greinacher A Clinician’s Perspective on Heparin-Induced Thrombocytopenia: Paradoxes, Myths, and Continuing Challenges Lawrence Rice.
Theodore E. Werkentin, Andreas Greinacher.