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eBook Genetic Polymorphisms and Susceptibility to Disease (Taylor Francis Series in Pharmaceutical Sciences) ePub

by Mark Steven Miller,Maureen Cronin

eBook Genetic Polymorphisms and Susceptibility to Disease (Taylor  Francis Series in Pharmaceutical Sciences) ePub
Author: Mark Steven Miller,Maureen Cronin
Language: English
ISBN: 0748408223
ISBN13: 978-0748408221
Publisher: CRC Press; 1 edition (April 27, 2000)
Pages: 288
Category: Medicine & Health Sciences
Subcategory: Other
Rating: 4.9
Votes: 388
Formats: lrf txt doc lit
ePub file: 1696 kb
Fb2 file: 1455 kb

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Series: Taylor & Francis Series in Pharmaceutical Sciences. I use this book to teach both pharmacists and pharmaceutical scientist graduate students

Series: Taylor & Francis Series in Pharmaceutical Sciences. I use this book to teach both pharmacists and pharmaceutical scientist graduate students. I think it is very good at identifying the barrier to drug transport in specific regions of the body as well as discussing technologies for delivery to those regions. I only wish the book was updated as well as the figures in color.

CARD15 and IL23R influences Crohn’s disease susceptibility but not disease phenotype in a Brazilian population. Inflamm Bowel Dis. 2008;14:674–9.

Inflamm Bowel Dis. 2008;14:204–12. CARD15 and IL23R influences Crohn’s disease susceptibility but not disease phenotype in a Brazilian population. 28. Borgiani P, Perricone C, Ciccacci C, Romano S, Novelli G, Biancone L, et al.

In subjects with genetic susceptibility factors, coronary artery disease is. .NT5E gene encodes for CD73 that transforms adenosine monophosphate (AMP) to adenosine, supporting a role for this metabolic pathway i.

In subjects with genetic susceptibility factors, coronary artery disease is determined by exposure to some risk factors. To date, more than 50 loci have been identified for susceptibility of coronary artery disease, as described by the meta-analysis carried out by the CARDIoGRAMplusC4D Consortium, on almost 185,000 cases and controls NT5E gene encodes for CD73 that transforms adenosine monophosphate (AMP) to adenosine, supporting a role for this metabolic pathway in inhibiting vascular calcification.

Genetic polymorphisms in these genes may result in partial or total loss of function and may increase the host's susceptibility to aspergillosis. Candidate gene association studies showed SNPs in SP-A2 and MBL significantly associate with patients of allergic bronchopulmonary aspergillosis and bronchial asthma with rhinitis. The patients carrying either one or both of GCT and AGG alleles of SP-A2 and patients with A allele at position 1011 of MBL had markedly higher eosinophilia, total IgE antibodies and lower FEV1 (the clinical markers of ABPA).

Susceptibility to infection by bacterium such as Bacillus anthracis has a genetic basis in mice and may also have a.

Susceptibility to infection by bacterium such as Bacillus anthracis has a genetic basis in mice and may also have a genetic basis in humans. In this study, we determined if polymorphisms in genes important in innate immunity are associated with increased susceptibility to infectious and non-infectious lung diseases, particularly tuberculosis and sarcoidosis, respectively, and therefore might be a risk factor for inhalation anthrax.

Genetic Polymorphisms and Susceptibity to Disease provides a reference for established researchers in genetic research. The book provides a broad but thorough overview of how allelic gene differences influence disease susceptibility in the human population, and will be a useful reference to researchers across a range of disciplines, for example, oncology, cardiology and immunology . In addition, this book serves as a primer for students first entering the field of genetic research.
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